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dc.contributor.authorMarques, Jorge
dc.contributor.authorDurán-Vila, Núria
dc.contributor.authorDaros, José A.
dc.date.accessioned2017-06-01T10:12:34Z
dc.date.available2017-06-01T10:12:34Z
dc.date.issued2011
dc.identifier.citationMarques, J., Duran-Vila, N., Daros, J.-A. (2011). The Mn-binding proteins of the photosystem II oxygen-evolving complex are decreased in date palms affected by brittle leaf disease. Plant Physiology and Biochemistry, 49(4), 388-394.
dc.identifier.issn0981-9428
dc.identifier.urihttp://hdl.handle.net/20.500.11939/5571
dc.description.abstractBrittle leaf disease or maladie des feuilles cassantes (MFC) is a disorder affecting date palms (Phoenix dactylifera L) which after a long declining process eventually leads to the death of the plant. No causal agent for the disease has been found so far but leaflets of affected palms are Mn-deficient despite the existence of adequate exchangeable Mn in the soils in which affected palms grow. The disease is specifically associated with an increase in a series of chloroplastic RNAs. A proteomic analysis of leaflets of affected and unaffected date palms showed differences in quantities of several proteins. Mn-binding PSBO and PSBP proteins, components of the oxygen-evolving complex of photosystem II, were decreased in affected tissue, reinforcing the relation between MFC and Mn deficiency. The quantities of other proteins were increased by disease suggesting a response to stress.
dc.language.isoen
dc.titleThe Mn-binding proteins of the photosystem II oxygen-evolving complex are decreased in date palms affected by brittle leaf disease
dc.typearticle
dc.authorAddressInstituto Valenciano de Investigaciones Agrarias (IVIA), Carretera CV-315, Km. 10’7, 46113 Moncada (Valencia), Españaes
dc.date.issuedFreeFormAPR 2011
dc.entidadIVIACentro de Protección Vegetal y Biotecnología
dc.identifier.doi10.1016/j.plaphy.2011.02.008
dc.journal.issueNumber4
dc.journal.titlePlant Physiology and Biochemistry
dc.journal.volumeNumber49
dc.page.final394
dc.page.initial388
dc.rights.accessRightsopenAccess
dc.source.typeImpreso


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